Retroversion of the uterus is a normal variation of female pelvic anatomy in which the body of the uterus is tipped toward the back rather than forward. It is commonly called a "tipped uterus."
Uterus retroversion; Malposition of the uterus; Tipped uterus
Retroversion of the uterus is common. It is the normal uterine position in about 20% of all women.
Weakening pelvic ligaments associated with menopause may cause this condition in women who previously did not have a retroverted uterus.
Enlargement of the uterus, either as the result of a pregnancy or a tumor, may also change lead to retroversion.
Scar tissue in the pelvix (pelvic adhesions) can also hold the uterus in a retroflexed position. Such scarring may result from:
Retroversion of the uterus almost never causes any symptoms.
Rarely, it may cause pain or discomfort.
A pelvic examination reveals the position of the uterus. However, a tipped uterus can sometimes be mistaken for a pelvic mass or an enlarging fibroid. A rectovaginal exam may be used to distinguish between a mass and a retroverted uterus.
An ultrasound examination can be used to determine the exact position of the uterus, if necessary.
Usually this condition does not cause problems.
Call your health care provider if you develop persistent pelvic pain or discomfort.
Atypical positioning of the uterus may be caused by endometriosis, salpingitis, or pressure from a growing tumor. These conditions should be ruled out in a patient with pain or other symptoms.
Treatment is usually not necessary. Any underlying disorders (such as endometriosis or adhesions) may be treated as needed.
There is no known prevention. However, early treatment of PID or endometriosis may reduce the chances of a change in the position of the uterus.
Lentz GM. Differential diagnosis of major gynecologic problems by age group: vaginal bleeding, pelvic pain, pelvic mass. In: Katz VL, Lentz GM, Lobo RA, Gershenson DM, eds. Comprehensive Gynecology. 5th ed. Philadelphia, Pa: Mosby Elsevier; 2007:chap 8.
Review Date: 10/28/2008
Reviewed By: Linda Vorvick, MD, Seattle Site Coordinator, Lecturer, Pathophysiology, MEDEX Northwest Division of Physician Assistant Studies, University of Washington School of Medicine; and Susan Storck, MD, FACOG, Chief, Eastside Department of Obstetrics and Gynecology, Group Health Cooperative of Puget Sound, Redmond, Washington ; Clinical Teaching Faculty, Department of Obstetrics and Gynecology, University of Washington School of Medicine. Also reviewed by David Zieve, MD, MHA, Medical Director, A.D.A.M., Inc.
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